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How Diet and Macros Shape Cholesterol, Liver Fat and Heart Disease Risk in Type 2 Diabetes — Independent of Weight Loss

How Diet and Macros Shape Cholesterol, Liver Fat and Heart Disease Risk in Type 2 Diabetes — Independent of Weight Loss

How Diet and Macros Shape Cholesterol, Liver Fat and Heart Disease Risk in Type 2 Diabetes — Independent of Weight Loss

Why Macros Matter More Than You Think, more than calorie-counting and calorie reduction

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Ultra-Processed Foods Dominate UK and US Diets: What It Means for Health and Policies

Ultra-Processed Foods Dominate UK and US Diets: What It Means for Health and Policies

Ultra-Processed Foods Dominate UK and US Diets: What It Means for Health and Policies

Ultra-processed foods (UPFs) have quietly taken over supermarket shelves and family dinner tables across the UK, the US, and much of the developed world. Once an occasional treat, these factory-made products, packed with additives, refined sugars, and industrial ingredients, now account for more than half of all food purchases in both countries. Health experts warn that this seismic shift in our diets is driving a surge in obesity, chronic disease, and early mortality, especially among the most vulnerable communities.

A must-read article.

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Unlocking the Secret to Longevity: Telomeres – The Blueprint for Cellular Health and Vitality
Inflammation, DNA, Mediterranean Diet, Oxidative Stress Olivier Sanchez Inflammation, DNA, Mediterranean Diet, Oxidative Stress Olivier Sanchez

Unlocking the Secret to Longevity: Telomeres – The Blueprint for Cellular Health and Vitality

The role of telomeres and targeted interventions

Over the past century, life expectancy has greatly increased in developed countries, but with it, chronic diseases such as cancer, cardiovascular disease, and neurodegenerative disorders also rise. Ageing processes, including telomere attrition (shortening of telomeres), lead to the deterioration of cell function, ultimately accelerating tissue degradation. Telomere shortening is a protective mechanism but paradoxically contributes to ageing and age-related diseases by triggering senescence or cell death once telomeres reach a critical length.

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